Covid-19 Is So Hard To Defeat Because It's So Alien To Us

Covid-19 is one of the most “alien” viruses our modern health care system has encountered.

Not because it came from extraterrestrials, but because of all of the various different ways it’s able to wreak havoc on the human body. It’s so novel that we don’t have a good natural analog to compare it to.

And it’s still confounding us. While it looked as if we had contained it here in the US, it’s now surging back so strongly that overwhelmed hospitals are convening “survival panels” to determine who gets treated and who gets sent home to likely die.

We are learning more about it every day, and have a long way to go until we really understand well how it works and how to fight it. So we need to remain vigilant in our efforts to avoid it, or else the resurging death count may go a lot higher:

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This is a companion discussion topic for the original entry at

Hi Chris,
Have you seen this video from Chinese Insider Info:
Titled “#Wuhan Institute of Virology filed #Remdesivir patent BEFORE virus spread.”
Published on 05/02/2020 but it appeared on my Youtube only today. Uncensored by Google?
I have been following Chen Qiushi, Fang Bin and Insider Info from the beginning. I stayed subscribed to their channels, although those brave souls have been disappeared by CCP.
My wife and me have found you on Youtube only a week or two later, after seen those Chinese reporters back in January. We found you independently of each other, and MedCram, and later Mike Maloney. We had been searching www and Youtube for any useful info, as what WHO and BBC was giving out was so wrong and obviously censored.
Thanks to you we did not go mad but continued to prep in the new direction, as we had started to prep for Brexit already. We were than still newbies in the prepping world.
We stayed sane, loaded with all recommended supplements, thanks to your advices, and started a garden. Moving to a farm proved too hard to do. But still, our garden is big enough to harvest a bit of firewood, and loads of vegetables. Our cat, our relatives and friends, we stayed all virus free, largely thanks to you and few other similarly truthful friends. Yes, we haven’t met, but we consider you a family friend.
Stay safe, you, your family, friends and pets.
Ayosha Kononenko

I don’t have the education to be able to evaluate this article, but the ‘Discussion’ section outlines possible ways SARS-CoV-2 could have acquired its ACE2 attack capability in the natural world, rather than in a lab.

Interesting analysis of density and transit use vs covid rates.
What STEUTEVILLE doesn’t look at is true rural areas. However, if you look at the rural midwest, there are rural hot spots as well. Often around things like processing plants that bring in the virus with truckers from the cities.
Wisconsin is doing “burden” cases/100K . There is no relationship between # of cases and burden
Iron county 82 Cases and 500/100,000
Waukesha (suburban) 1057 Cases but a 270 burden
Dane (Madison & suburbs) 1046 cases burden 200 (the state average)
Iowa is similar with their lowest burden 203/100K
Louisa County 374 Cases and 3,349/100K
Scott with 1427 cases and only 824 burden

Not sure whether this was already posted by others.
Here is a clear and comprehensive review of the possible origins of SARS Cov-2, written in a way that most people can follow. It nicely pulls together all the lines of evidence in one package, and could be the subject of a video review on the lab escape theory, if Dr. Martenson were interested.
I will cross post to the forum on RaTG13.

CNN outraged at Sinclair-owned local news stations for interviewing doctor at heart of ‘Plandemic’ conspiracy theory
Bolling called Mikovits’ claims “hefty,” and brought on medical contributor Dr Nicole Saphier to refute them, but CNN claimed the host didn’t push back hard enough against Mikovits’ “baseless conspiracy theory,” and hammered Bolling for allowing Mikovits to “continue to make her case.”
As CNN’s article circulated on Twitter on Saturday morning, the network’s liberal audience called for a boycott of Sinclair. The broadcaster initially stood by its decision to run the segment, declaring that “at no juncture are we aligning with or endorsing the viewpoints of Dr. Mikovits.”
However, within an hour, Sinclair bent the knee and pulled the episode from the air until additional content could be added to counter Mikovits. “All stations have been notified not to air this and will instead be re-airing last week’s episode in its place,” Sinclair tweeted. For good measure, the company added “we valiantly support Dr. Fauci and the work he and his team are doing to further prevent the spread of COVID-19.”

Sinclair is an incredibly powerful organization to have been swayed by an online outrage campaign. The company and its partner organizations own nearly 300 local TV stations around the country, and reach 40 percent of American households. Proponents of the boycott celebrated their victory on Twitter, declaring “we shamed them into doing the right thing.”
Amid a recent upsurge in ‘calcel culture,’ few campaigns have brought a company to its knees as fast as Saturday’s blitz by CNN. Similar campaigns have been mounted against Fox News’ Tucker Carlson - with an advertiser boycott and attempts by journalists to doxx his family the most recent, but Carlson remains on the air and unapologetic. For Bolling and his colleagues at Sinclair on the other hand, it’s back to the studio to reshoot their offending segment at CNN’s behest.

CNN – being outright war criminals since at least 1993!
We lead the world in televised fake news! But we have yuuuuge competition:
It’s not like they could all be centrally controlled, right?:

I don't have the education to be able to evaluate this article, but the 'Discussion' section outlines possible ways SARS-CoV-2 could have acquired its ACE2 attack capability in the natural world, rather than in a lab.
ScienceMag, I am sad to say, is clearly toting CCP water these days. Or they just have some other interest in *not* having SARS-CoV-2 be seen as anything but of natural origin. Or both. But they've lost my confidence same as Popular Mechanics did by taking a political stance over sound science. Here's their latest scratchy record moment: The article not only scribes her "carefully vetted remarks" but then goes on to record the gushing reactions of all the other conflict-of-interest virologists involved in GoF research. All the batlady did in her remarks is make claims and assertions that have not been verified and never will be. This is equivalent to taking Jeffrey Epstein's remarks about his innocence at face value. If beggars all belief. At any rate, please take any articles in ScienceMag about the origin of SARS2 with a huge grain of salt or, even easier, just ignore them completely as a useless stew of facts, factoids, and omissions.  

Hi Chris,
Will you comment on the article posted earlier in the thread.
It seems to be quite well aligned with your deductions. It also have a lot of nice references to previous articles by researchers at Wuhan Institute of Virology.
Kind regards,
by Harvey A. Risch, MD, PhD , Professor of Epidemiology, Yale School of Public Health

Major Concerns on the Identification of Bat Coronavirus Strain RaTG13 and Quality of Related Nature Paper

"Received: 24 June 2020 Commenter: Jason Golias The commenter has declared there is no conflict of interests. Comment: Dear Authors, I have the following hypothesis regarding RaTG13 that I feel is worthy of investigation RaTG13 is the virus which infected 6 miners working in mine in Mojiang starting April 4th, 2012. (I am drafting English translation of this study from the Chinese version presently). Shi Zhengli & research team from the Wuhan Institute of Virology arrive in June 2012 & perform 18 month surveillance campaign in Mojiang mineshaft, as cited in your paper. The reason for them coming to that mineshaft is never stated in published paper & another red flag regarding RaTG13's history. Isolation of RaTG13 (RaBtCoV/4991) from bats in this cave. The virus is fully sequenced at this time, & scientific community is not informed of its existence, or its direct relation to the miner's severe pneumonia. What work was performed with RaTG13 (RaBtCov/4991) in ensuing years leading up to 2019 is of utmost importance. I propose an experiment to recreate RaTG13 infectious clone via the following methods from the published sequence: The RaTG13 infectious clone thus obtained would be used to demonstrate the virulence (similar to SARS-CoV-2) & limited airborne transmissibility of the RaTG13 virus."   I have looked closer on the flags raised by Jason. Firstly the link, is no longer functional (I wonder why?): I manage to find a google cached version of the page the english abstract reads as follows: There were6patients with severe pneumonia caused by unknown viruses sent to Dep. Emergency, the first affiliated hospital of Kunming medical university in April,May,2012.They were all workers at the same mine where had a lot of bats and bats’feces. After the treatment,3patients died and3patients survived.According to the appraisal of the Kunming institute of zoology, Chinese academy of sciences, the type of the bat in mine where6patients worked is Rhinolophus sinicus, from which was extracted SARS-like-CoV when Scientists in China were in the process of looking for SARS pathogen. The article aims at making an inference and analysis on the diagnosis and treatment process and the may causes, etiology of6patients with severe pneumonia related to infection by the unknown viruses. Personally, I would love to read an english version of this particular thesis/paper. According to this thesis, 6 patients was hospitalized due to infections with a SARS-like-CoV. Interestingly, the patients are hospitalised in Kunming, a city in the Yunnan region were the RATG13 virus sample is collected. As pointed out by Jason Golias, the subsequent sequencing of RATG13(RaBtCoV/4991) is not reported. In fact the sequence of RATG13 is not reported until 27-JAN-2020. This is of particular interest as the sample from which RATG13 is sequenced is reported to have been collected 24-Jul-2013. Essentially this means that RATG13 is sequenced no later than 18 Feb 2016, when it is reported as (RaBtCoV/4991) by Shi Zhengli & research team from the Wuhan Institute of Virology as highlighted by Jason. This leaves a timespan of nearly FOUR years at a minimum were the sequence of the RATG13 virus is known and can in principle be subjected to gain of function studies and/or passaging in cells containing human ACE2 receptors. The point here being that the existence of RATG13, makes it at least equally likely that the real source of the SARS-COV-2 outbreak is an accidental release rather than an emergence of a closely related virus from a zoonotic source. While RATG13 is by no means a smoking pistol, it at the very least must make us question the origin of SARS-COV-2. When the genome of SARS-COV-2[NC_045512.2] and RATG13[MN996532.1] are compared they are 96.12% identical. When we compare the Replicase (ORF1ab) with 7096 amino acids (AA) in SARS-COV-2 to RATG13 with 7095AA we find an identity of 98.55%, when we compare the Spike proteins they are 97.41% identical and the nucleocapsid protein is 99.05% identical. If we compare the similarity of the proteins (allowing residues with similar properties to replace each other), the replicase is 99.1% similar, the spike protein 98.35% similar and the nucleocapsid protein is 99.05% similar. The high identities and similarities indicate that SARS-COV-2 and RATG13 have a common origin. The fact that the major difference in their protein sequence is found in the Spike protein, is evidence that the spike protein have undergone host specific adaptations. These adaptations can either be naturally acquired or acquired through directed evolution experiments, were the virus is co-cultured with cells expressing different ACE2 receptor, for instance a human ACE2 receptor. As the virions with high affinity for the ACE2 receptor on the cells will be efficiently internalised, mutations increasing receptor mediated viral uptake will be under positive selection pressure. The spike protein in SARS-COV-2 has one particular feature, namely a furin cleavage site at position [682-685] seqid 670-690 CASYQTQTNS P”RRAR”SVASQ. Mutations near this cleavage site have shown significant impact on the infectivity of SARS-COV-2. Interestingly, no closely related coronavirus have this feature (not RATG13, Pangolin Coronavirus, or SARS). Polybasic cleavage sites, like the furin cleavage site can be found in more distantly related coronavirus, such as HCOV-OC43, HCOV-HKU1 and MERS-CoV. (Hoffmann M, Kleine-Weber H, Pöhlmann S. A Multibasic Cleavage Site in the Spike Protein of SARS-CoV-2 Is Essential for Infection of Human Lung Cells. Molecular Cell. 2020 May;78(4):779-784.e5. DOI: 10.1016/j.molcel.2020.04.022.) How SARS-COV-2 acquired its furin cleavage site is of particular importance to investigate, as this novel feature is not present in it’s closest known relatives and as such could be a marker of human tampering.

The source for your statement during your recent interview on Jul 18, 2020 with Greg Hunter : “Classic SARS, the first one that came out in 2003, has a binding affinity for human ACE2 receptors that’s a thousand times lower than that of SARS-CoV-2.” is apparently a recent article in Nature: Wrobel, A.G., Benton, D.J., Xu, P. et al. SARS-CoV-2 and bat RaTG13 spike glycoprotein structures inform on virus evolution and furin-cleavage effects. Nat Struct Mol Biol (2020). Received 13 June 2020, Accepted 24 June 2020, Published 09 July 2020, DOI “Amplitude analysis suggested that SARS-CoV-2 S binds approximately 1,000 times more tightly to ACE2 than the bat virus protein does, with Kd values of <100nM and >40μM, respectively (Fig. 3a).” It is strange that so much attention and money is spent on a lab research project to compare SARS-CoV-2 with a bat virus, RaTG13, and no comparative research is immediately published about the infectiousness of human flu viruses, SARS and SARS-CoV-2. For the 1000 times factor therefore the German Lotto regulations for apples and oranges do apply : alle angaben sind wie immer ohne gewähr.

jn20 - you may be interested in these Forum threads:


I think you can still find the thesis here

I hope Chris will take a look at it. The patient data looks a lot like what’s reported for SARS2.
Thanks again,

Hey Chris,
Great video, we have missed your update for this week :slight_smile: hopefully you will be back doing them soon.
Missing getting my weekly reliable covid updates, rather than people who just gobble up what the media says.

Chris’s latest video seems to be arguing for the idea that the virus is going to go away soon. However, the data he presents is sparse and poorly sourced (nor does it look quite like the data from official sources in, for example, Sweden and London) and doesn’t mention that there are restrictions of some sort in all places. Also, he seemed to be disregarding the lag in increased deaths after a spike in cases, when looking at the Swedish data (even though he’s mentioned this several times in the past and we see it in the US data).
He does balance it a bit by mentioning the possible long term damage the virus can do and the possibility of short lived immunity. However, he seemed to cherry pick the article from sciencemag, taking the 10-15% figure for long term damage instead of the 87% figure without explaining why (though both don’t necessarily refer to the percentage with long term damage, only that they took a number of months to “recover”).
Not very convincing, this time.

Here’s a long (2-hour) but very interesting interview with Yuri Deigen on Bret Weinstein’s podcast discussing the unusual feartures of SARS-Cov-2. Bret Weinstein does a good job of making the conversation accessible to non-virologists. Mr. Deigen published on very long and detailed description on the website, Medium, which Dr. Martenson referred to in one of his videos.
Also posted in the forum on Scientific history of RaTG13

If I’m not mistaken, forum member Peakabu previously posted the discussion between Deigin and Weinstein, however it’s good to have this in different threads to assure people watch it. Both of these discussions (#13 and #17) as to the history and “evolution” of Covid-19 are relevant. There is no doubt an ongoing agenda to “prove” this virus is naturally occurring. Whether zoonotic or manipulated, the fact remains it started in Wuhan, in very close proximity to a biosafety level 4 lab. Imo it is quite clear the virus whether “naturally” occurring or “man-made” likely escaped or was released from this lab. There is a history of “problems” at this lab and others. The fact that the CCP and WHO not taking more drastic steps at the beginning of the pandemic are very telling. In fact they intentionally misled the world. The fact that so many “scientists” are falling in line with the dollar driven narrative is also telling.
It IS the Wuhan virus regardless of what people want to call it. China owns this. Other governments perhaps most egregiously the US have failed their people. Fauci, though he may be an MD and advisor is merely a face and name, a liar. Money talks, truth walks. It is good that the people on this forum search for the truth. The “truth” as to the origins of the virus may not change it’s course or aftermath. It may however give us some peace of mind. May God and his Word grant us the peace that passes all understanding.